Cannabinoids in Chronic Pain: Therapeutic Potential Through Microglia Modulation
Van Den Hoogen, Nynke J., Department Of Physiology, Pharmacology, University Of Calgary, Department Of Comparative Biology, Experimental Medicine, Hotchkiss Brain Institute, Harding, Erika K.
2-AG activates both CB1R and CB2R. Pain 162, S1–S2. Pain 154, 160–174. Activation of dorsal horn cannabinoid CB2R receptor suppresses the expression of P2Y12 and P2Y13 receptors in neuropathic pain rats. Pain 162, S26–S44.
Tags:
- Cannabinoid receptor type 2
- Endocannabinoid system
- Microglia
- Cannabinoid receptor
- Pain
- Chronic pain
- Medical cannabis
- Cannabinoid
- Neuron
- Pain management
- Inflammation
- Neuropathic pain
- Neuroscience
- Neurochemistry
- Physiology
- Neurophysiology
- Cell biology
- Biochemistry
- Cell signaling
- Signal transduction
- Cell communication
- Biotechnology