Cannabinoid Compounds as a Pharmacotherapeutic Option for the Treatment of Non-Cancer Skin Diseases
Robert Ramer | Burkhard Hinz
PPARγ was found to be activated by THC [76] and CBD [77]. In addition to the two cannabinoid receptors, human keratinocytes have also been shown to express TRPV1 [55]. Human keratinocytes have also been shown to express PPARα/γ/δ receptors and GPR55 [55]. Regulation of the Endocannabinoid System in Skin Diseases 3.3.1. In addition, AEA and 2-AG have been reported to be reduced in keratinocytes from psoriasis patients, while PEA levels are increased [7].
Tags:
- Cannabinoid
- Anandamide
- Cannabinoid receptor 2
- Psoriasis
- Interleukin 17
- Melanocyte
- Fatty-acid amide hydrolase 1
- Endocannabinoid system
- Interleukin 23
- 2-Arachidonoylglycerol
- Cannabinoid receptor 1
- Cannabinol
- Cannabinoid receptor
- Keratinocyte
- Dermatitis
- Atopic dermatitis
- Tetrahydrocannabivarin
- TRPV1
- Wound healing
- Anti-inflammatory
- Janus kinase
- Tumor necrosis factor
- Inflammation
- 2-Arachidonyl glyceryl ether
- Epidermis
- Immunosuppressive drug
- Itch
- Interleukin 6
- Tetrahydrocannabinol
- Sebaceous gland
- Pattern hair loss
- Allergy
- Healing
- Macrophage
- Neurochemistry
- Cell biology
- Biochemistry